Alcohol use has complex effects on cardiovascular health (CV). The association between drinking disorders and CVs such as high blood pressure, heart disease, stroke, cardiovascular disease, and cardiomyopathy have been extensively studied and described in this review. Although many behavioral, genetic, and biological factors contribute to the link between alcohol use and CV disease, dose, and pattern of alcohol consumption seems to justify this significantly. Low to moderate alcohol consumption can reduce certain mechanisms such as risk and hemostatic factors affecting atherosclerosis and inflammation, pathophysiologic processes involving many CV diseases. But any good drinking habits should be weighed against the negative effects of the body, including mitochondrial dysfunction and changes in blood circulation, inflammatory response, oxidative stress, and systemic cell death, and anatomical damage to the CV system, especially the heart. itself. Both the negative effects and the positive effects of alcohol use on certain CV cases are presented here. The review concludes by suggesting several promising future research methods related to alcohol use and CV disease. This includes using specific alcohol biomarkers to ensure your reporting of alcohol levels; to study the potential mediation of various genetic, social, and economic factors, and races and ethnicities that may affect alcohol use and CV diseases; reviewing the interaction of alcohol drugs in heart patients; and evaluating CV effects on alcohol use in adults and the elderly.
This study provides evidence of a causal link between excessive drinking and an increased risk of stroke and heart disease. The causative role of alcohol in some heart diseases requires further research. Mendelian randomization (MR) is an epidemiological procedure that uses genetically modified genes to determine their role in disease risk. MR studies are less vulnerable to bias based on confusion, retransmission, and error rate compared to standard observational studies. In a previous study of MR in European ancestry, an increase in single-nucleotide polymorphism caused by alcohol in the ADH1B gene was associated with a higher risk of heart disease and stroke, which contradicted the findings.3-6 Results from a recent MR study conducted in Chinese people showed that drinking alcohol of 2 alcohol-related SNPs is associated with a higher risk of ischemic stroke and internal bleeding but not associated with myocardial infarction. Meta-association analysis of wide-spectrum genomes identified multiple SNPs associated with alcohol use. Using these SNPs as mutants, conduct an MR study to investigate the relationship between alcohol use and CVDs, including stroke, stroke, atrial fibrillation, heart failure, venous thromboembolism, vascular disease, aortic valve. stenosis, and aortic aneurysm. In the second analysis, the associations of expected alcohol use and potential mediators (blood pressure and serum lipids) and the confusion (smoking and education) of alcohol-CVD organizations were assessed.
Using alcohol increases the risk of CVD high blood pressure. A meta-analysis of randomized controlled trials showed that reducing alcohol consumption reduced blood pressure in response to daily alcohol consumption. genetic predisposition to alcohol use and ADH1B mutations. High-impact drinking is associated with an increased risk of high serum cholesterol and triglyceride levels. low levels of fibrinogen. UK Biobank data analysis was performed under request 29202. Summary statistical data on genetic disorders of stroke, heart disease, and atrial fibrillation were provided by the MEGASTROKE Consortium, Coronary Artery Disease Genome-Wide Replication and Meta-Analysis and Coronary MEGASTROKE project financial resources specified.
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