Magnesium can be used in the treatment of various diseases which include:
PMS (premenstrual syndrome): Magnesium consumption may affect premenstrual syndrome (PMS). Magnesium supplementation, coupled with vitamin B-6, has been demonstrated in small-scale trials to help alleviate PMS symptoms. In addition, it also evidenced that magnesium supplements help to avoid bloating, mood swings, and breast discomfort associated with PMS.
Headaches caused by migraines: Magnesium can help to alleviate or prevent headaches. The explanation is that magnesium deficiency can trigger migraines by affecting neurotransmitters and causing blood vessel constriction. It is conceivable that migraineurs’ blood and tissues have varying magnesium levels. People who suffer from migraines may have low magnesium levels in their brains.
Anxiety: Magnesium levels may have a role in depression and anxiety. Low magnesium levels lead to an increased risk of anxiety in several systematic evaluations. It can be explained in part by activity in the hypothalamic-pituitary-adrenal (HPA) axis. HPAs regulate how humans react when they are stressed. To establish how effectively magnesium supplements help to decrease anxiety, researchers must undertake high-quality trials.
There are several ongoing and existing research studies which claim that magnesium supplements do not decrease blood pressure to a considerable extent. However, a thorough investigation would be required to discover how magnesium may be utilized to prevent heart disease and blood clots.
Magnesium as a Cofactor
Magnesium is abundant in cells and catalyzes more than 300 enzyme processes. It is essential for its crucial counteractions to the energy-yielding ATP and nucleic acids. In addition, many adenosine triphosphate ATP-generating processes require the presence of magnesium to function efficiently. ATP is necessary for numerous activities, including glucose consumption, protein synthesis, nucleic acid, and coenzyme synthesis, muscular contraction, methyl group transfer, and similar activities. Hence, any alterations in the magnesium metabolism would hamper these functions. It is critical to maintain adequate levels of magnesium, as it influences ATP metabolism, muscular functions, neuronal activities, and neurotransmitter release. The calcium-activated ATPase of the sarcoplasmic reticulum facilitates the reuptake of calcium by magnesium during muscle contraction. Magnesium is also involved in signal transduction pathways, cell proliferation, cell adhesion, and serves as a transport component of transmembranes. Acetylcholine released at motor endplates is antagonized by magnesium. In addition, magnesium inhibits calcium-induced cell death. By inhibiting apoptosis triggered by mitochondrial permeability transitions, it blocks calcium-induced apoptosis.
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